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Of note, our previous work demonstrated that the gut metabolite NaB markedly reversed HFD-induced dysbiosis of the gut microbiota in mice and significantly elevated the abundance of Lactobacilli in the gut 10 , which supported the hypothesis that NaB might have the potential to improve GLP-1 resistance

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IDE knockout mice are both glucose-intolerant and hyperinsulinemic, supporting the concept that IDE is important in the maintenance of normal blood glucose and insulin levels [2]

Baseline bloodwork (especially IGF-1) documents the starting point and how much room there is to improve