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The neurotoxic effects of mHtt are mediated by the induction of mitochondrial defects, which in turn result in aberrant energy metabolism and HD-related neuronal dysfunction ( Studies have demonstrated that mHTT interacts with the PGC-1 promoter, directly impairs the activity of PGC-1, and impedes the capacity of PGC-1 to open downstream target genes (Hu et al., 2025)

An early work, presented in 1995 by Dentico and colleagues, evaluated the effect of 30-day administration of high doses of intravenous or intramuscular GSH on liver cytolysis indexes in patients with chronic steatosic liver disease (77)

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