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In pathological conditions, astrocytes undergo significant molecular and morphological remodeling, adopting either a detrimental A1 (neurotoxic) or beneficial A2 (neuroprotective) reactive state that, respectively, drives injury progression or promotes tissue repair [132, 133]

They proposed that NOX2 and NOX4 play a crucial role in the development of IIM, possibly by depleting NADPH to generate excessive ROS, leading to oxidative damage to tissue cells and inducing ferroptosis

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