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Stopping the medication abruptly can trigger its own set of issues, including potential weight regain and another round of metabolic disruption

MT-2s cyclic structure confers significantly enhanced resistance to enzymatic degradation, with an effective research half-life of approximately one to two hours, providing a substantially wider in vitro and pre-clinical experimental window than the endogenous compound

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Inhibiting GPX4 expression in hepatocytes exacerbates mitochondrial damage and lipid peroxidation, further driving ferroptosis and worsening the SLI (Wang et al., 2023b)