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Apoptotic bodies of bone marrow mesenchymal stem cells inhibit endometrial stromal cell fibrosis by mediating the Wnt/-catenin signaling pathway [J]

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In the 1980s, Holers theorized that the complement system might be activated not just during infection but with other diseases, such as arthritis, lupus, lung disease and Alzheimer's

These effects are achieved through the binding of propionate and butyrate to GPR41, GPR43, and GPR109A receptors, as well as through the inhibition of histone deacetylase (Cho and Shore, 2016)