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37 In our study, we found that Clopidogrel downregulated the expression of phosphorylated eNOS and induced ER stress in the rat gastric mucosa cells and the BPC 157 protected against Clopidogrel-induced gastric mucosa dysfunction by alleviating ER stress and promoting eNOS -mediated angiogenesis whereas the effect of L-NAME is completely the opposite
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They contain the same active ingredient semaglutide, tirzepatide or orforglipron but add minor but clinically important modifications such as using a different salt form, adding different inactive ingredients and varying drug concentrations or dosages
This post-translational modification inhibits PDIs catalytic activity, impairing its role in protein folding
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