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Under physiological conditions, GSH quenches oxidizing substances (such as reactive hydroxyl free radicals, peroxynitrite, and H 2 O 2 ) directly or reduces hydrogen peroxide (or lipid peroxide (lipid-OOH)) to water (or the corresponding lipid alcohol (lipid-OH)) under the catalysis of GPX (summarized in Fig

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Analysis of single-cell sequencing datasets, along with immunohistochemistry and immunofluorescence staining validation, have revealed that abnormally activated lipid peroxidation in CD163+ macrophages and CD10 + PC+ (pyruvate carboxylase) epithelial cells suggests their potential involvement in ferroptosis within the glomerular compartment [132]