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Results Semaglutide attenuates LPS-induced neuroinflammation To investigate how GLP-1 receptor activation modulates inflammatory responses in the brain, we first established a model system allowing detailed temporal analysis of neuroinflammatory processes using LPS

Engineered AAVs for efficient noninvasive gene delivery to the central and peripheral nervous systems

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Ferroptosis fundamentally differs from conventional cell death mechanisms such as apoptosis, necrosis, and autophagy