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Disruptions in choline metabolism, whether due to impaired uptake or blockade of de novo PC synthesis by pharmacological inhibition of ChoK or CTP:phosphocholine cytidylyltransferase alpha (CCT), alter the cellular phospholipid pool composition, the integrity of the mitochondrial membrane, and the overall cellular activity, leading to immune dysfunction and dysregulated inflammation across species (67, 6972)

Lee 2014

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Once activated, the mentioned pathways encourage growth hormone release, which in turn modulates downstream signals, insulin-like growth factor 1 (IGF-1), and other metabolic factors