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we therefore report start-of-window risk sets

The mitochondrial origin connects MOTS-c directly to cellular energy metabolism and positions it as an endogenous regulator of metabolic function

Nonetheless, considering the clinical relevance of GLP-1 therapies and their potential impact on energy balance beyond food intake and body weight, future studies should investigate whether GLP-1 agonists influence hypothalamic neuropeptide expression or signaling cascades in MC4R-deficient models