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-Amyloid peptides enhance -synuclein accumulation and neuronal deficits in a transgenic mouse model linking Alzheimers disease and Parkinsons disease

But it also raises legitimate questions about potency and quality control that patients should not dismiss

Millman, [email protected] Disclaimer All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers

The GLP-1s have multi-organ protective therapies with benefits spanning cardiovascular, kidney, liver, metabolic, and emerging neurologic domains as well