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Regulation of potassium export channels by GSH is not yet proven in LAB and the mechanism for protection is suggested to be either sacrificial action of GSH which prevents rapid fall of intracellular pH or glutathionylation of glyceraldehyde 3-phosphate dehydrogenase (GAPDH) enzyme which helps to sustain glycolysis at the required level

By suppressing caspase activation and modifying Bcl-2 family proteins, melatonin also promotes cellular viability