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The two compounds activate distinct somatotroph receptors (GHRH-R and GHS-R1a), producing additive GH release in published rodent and primate models without the cortisol or prolactin spillover seen with earlier GHRPs, because they couple to different intracellular signalling cascades

This increase in intestinal gluconeogenesis leads to portal glucose sensing to regulate energy homeostasis and improve insulin sensitivity 58

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