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ER stress and basement membrane defects combine to cause glomerular and tubular renal disease resulting from Col4a1 mutations in mice

To achieve this, it will be necessary to complement chemical studies with more comprehensive biological evaluations, including more complex cell models, detailed mechanistic studies, pharmacokinetic analyses, and in vivo assessments

Consequently, they may exhibit undefined or broad specificity and relatively high equilibrium dissociation constant (Kd) values for their targets [442]

Pharmacogenomics: Advances in pharmacogenomics may eventually allow for formulations more precisely tailored to individual genetic profiles