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These include, increased inducible nitric oxide (NO) synthase (iNOS)-derived NO production [39, 40], increased inflammation [30, 39], increased oxidative stress [30, 31, 38], and increased apoptosis and -cell death [39, 40]

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4144 Moreover, it has been shown that GLP-1RAs also promote satiety trough the activation of the nucleus accumbens by increasing glutamatergic signaling, thus suppressing feeding behavior

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