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Chillaxcine works on three separate pathways of the stress response simultaneously

Increased cell death may be induced by an increased oxidative stress burden or a modification of the redox balance of gastric epithelial cells following Gln depletion 15,47

Thus, the identity of ACs, together with the upregulated expression of receptors such as UCP2, orchestrates immunometabolic states that limit phagocyte plasticity and prioritize inflammatory resolution and tissue protection over antibacterial defense [82]