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Early animal experiments with hypothalamic lesions and post-mortem examinations in humans with morbid obesity led to a proposal of the dual center hypothesis, postulating that ventromedial nuclei (VMN) serve as the satiety center and the lateral hypothalamic area (LHA) - as the feeding or hunger center (4) that when stimulated results in hyperphagia and subsequently hypothalamic obesity (5, 6)
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Thyroid ultrasound revealed heterogeneous echotexture with increased vascularity (158)
Tirzepatide treatment is fundamentally different
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