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At the same time, the elevation of p-CREB induced by BHBA was also decreased in KN-62-treated neurons, suggesting that the BHBA-induced rise of p-CREB levels depends on the activation of Ca 2+ /CaM/CaMKII (Figure 4B)

co-administration studies remain a relatively small subset

The clinical impact of dual agonism will ultimately depend on addressing current limitations, including cost, access, and long-term safety questions

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