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Such a notion of receptor activation was supported by previous mutagenesis studies, which showed that individual mutations of residues F279 6.51 , F312 7.42 , and W276 6.48 in the aromatic cluster significantly lowered the potency of ghrelin in activating GHSR 34,37

& Yasuhara, M

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GPX4 is required for repression of both canonical CASP1- and non-canonical CASP11-mediated cleavage of GSDMD 56 , indicating that GSH-dependent antioxidant systems do not only inhibit inflammasome assembly but also limit the oxidized membrane environment that favors gasdermin pore formation