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GLP-1 receptor activation in the brain's appetite control centers promotes satiety and reduces caloric intake, while GIP receptor engagement supports optimal energy balance

Hypoxic conditions elevated the inflammation in steatotic hepatocytes and increased the release of EVs derived from fat-laden hepatocytes, which stimulate the activation of hepatic stellate cells and the inflammatory phenotype in Kupffer cells [111, 112]

Lyophilization helps preserve peptide stability during storage while allowing controlled reconstitution for laboratory experimentation

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