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These peripheral and central deficits can include severe gastrointestinal lesions, intracranial (superior sagittal sinus) hypertension, brain swelling and lesions, portal and caval hypertension, aortic hypotension, peripheral and central thrombosis, inferior caval vein and superior mesenteric vein congestion, azygos vein failure, electrocardiogram (ECG) disturbances, and heart, lung, liver, and kidney lesions (Vukojevic et al., 2018

T-cell responses during experimental colitis are independent of H4R, histamine, or MC-derived histamine The pathogenesis and severity of Ox or DSS experimental colitis is often attributed to adaptive immunity, in particular the T-cell response

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