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First, although we identified significant alterations in pathways such as fatty acid -oxidation and PPAR signaling, we lacked direct functional validation through experiments such as mitochondrial respiration assays, gene expression analysis of key regulators (e.g., PPAR target genes), or isotopic tracing of fatty acid oxidation

GLP1s in higher amounts also act on portions of the brain that suppress appetite and signal satiety (feeling full)

Together, these discoveries firmly establish CoQ 10 oxidoreductases as a critical antiferroptotic defense system

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