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By employing whole-genome haploid screening and CRISPR-Cas9 technology, two pivotal drivers of ferroptosis were identified: Acyl-CoA synthetase long-chain family member 4 (ACSL4) and lysophosphatidylcholine acyltransferase 3 (LPCAT3), both of which function as membrane remodeling enzymes [75,76,77]

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In the insulin-resistant state in both humans and animals, abnormalities in insulin-induced NO synthesis via the IRS/PI3K/Akt/eNOS pathway may contribute to altered vascular function and hypertension (102,119,120,168)