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By redirecting electron flux and preventing excessive reduction of the respiratory chain, AOX mitigates ROS overproduction while maintaining redox balance and metabolic continuity particularly under drought, salinity, or temperature extremes

The kinetic data can be best explained by a ternary complex in which PfGrx recruits and activates GSH at the 1st glutathione-interaction site to facilitate the rate-limiting glutathionylation of roGFP2 WT (S 2 ) and its subsequent deglutathionylation in accordance with monothiol mechanism (iii) from Fig

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