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This continuous feedback loop supports dynamic modifications in drug release profiles, surface chemistry, or dosing schedules, ensuring that treatments evolve in step with disease progression and individual patient responses

In KRAS-driven NSCLC, LDHB has been identified as a key regulator of GSH-dependent ferroptosis resistance through STAT1 signaling, although the precise molecular mechanisms underlying LDHB-mediated STAT1 activation remain incompletely elucidated (49)

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The authors concluded that deacetylation occurs rapidly and completely before or immediately upon entry into systemic circulation indicating that the full administered dose is converted to GSH prior to reaching the bloodstream