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Conclusion In this comprehensive review, we set out to unravel the efficacy of nutritional strategies in mitigating effects of ES on later-life behavioral outcomes across clinical- and preclinical literature

IL-15-mediated downregulation of FOXO1 and upregulation of CXCR6 increases the sensitivity of these cells to metabolic cues such as acetate and extracellular ATP, promoting MHC class I-independent killing of hepatocytes (142)

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A study reported that retinoid acid can cause significant trans-epidermal water loss (TEWLs) that can further impair skin barrier and induce more skin irritation [3]