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T2D attenuation

Combined with the structural changes to the Purkinje cells [179, 180], possible Purkinje cell loss [181], Purkinje cell axonal alterations, and/or abnormal Purkinje cell synaptic organization [182, 183] reported in postmortem human ET tissue, the VIM neurons are likely entrained by abnormal cerebellar activity

This shift has been facilitated by significant technological and methodological advancements that hold great promise for advancing PD research and bridging the existing gap in disease-modifying therapeutics that are personalized to the needs of each patient