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Sulfasalazine acts as a competitive inhibitor of SLC7A11 by interacting with the backbone of TM1a and R396 as well as Y240, Y244, and Y444 of SLC7A11 [203]

Introduction: Metabolic dysfunction-associated steatotic liver disease (MASLD) affects approximately 25% of the global population [1] , and 20-30% of these cases progress to a more severe stage known as MASH [2]

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Overload of mitochondria with ammonium instigates over-production of reactive oxygen species and opening of mitochondrial permeability transition pore, which further damages astrocytes and induce astrocyte swelling