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In this study, we showed that systemic MOTS-c treatment in two different pathophysiological mechanisms of diabetes in mouse models (-cell senescence in NOD mice and insulin-antagonist S961-induced insulin-resistant C57BL/6 mice) can reduce senescence in pancreatic -cells and delay the onset of diabetes
Schnemann, B
While GLP-1 and GIP regulate satiety, metabolism, and weight loss, NPY2 receptor agonism introduces a novel mechanism aimed at modulating central hunger signaling, potentially addressing both the biological and behavioral drivers of obesity
Note: if you are suffering from a medical condition it might be that you can receive this treatment from your NHS GP
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