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(Research has identified mast cells as one of the immune cell types involved in the inflammatory cascades that influence arterial plaque development and lipid-related cardiovascular risk, as I note in my MCAS info page.) In neurological disease , research in animal models of stroke and vascular dementia has shown that GLP-1RAs reduce both microglial activation and neuroinflammation, leading to better protection of the brain from injury and cognitive deterioration

While glutathione therapy shows promise, its important to approach it as part of a balanced health plan

Studies in FXR-null mice further indicate that loss of FXR disrupts glutamatergic, GABAergic, serotonergic, and noradrenergic neurotransmission in the hippocampus and cerebellum, resulting in impaired cognitive performance and motor coordination [92]

The right answer is not always the GLP-1