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Notably, considerable metabolic improvement was achieved at relatively low doses of GLP1/E, thus increasing the safety margin of this compound, which did not cause uterotrophic effects per se

These findings indicate potential pathways through which incretin therapy may: Stimulate bone formation by activating osteoblasts Inhibit bone resorption via osteoclast suppression Influence calcium and phosphate homeostasis Together, these mechanisms offer biologic plausibility that GLP1 RAs could help preserve bone strength

Synergistic Detoxification: Fasting is a natural "reset" for the liver

Carnitine/acylcarnitine Translocase and Carnitine Palmitoyltransferase 2 Form a Complex in the Inner Mitochondrial Membrane