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By studying NAPQI rather than acetaminophen, we were able to focus exclusively on genes and pathways related to the metabolism and toxicity of this highly reactive acetaminophen metabolite rather than its metabolic generation, which is subject to variation as a result of interindividual differences in sulfation, glucuronidation, and the activity of CYP enzymes

Prognostic biomarkers for esophageal adenocarcinoma identified by analysis of tumor transcriptome

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Dietary and nutritional supplements can help address this gap until innovative therapeutic methods are established