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Caspase inhibition lowered serum ALT in the short term but may have redirected cells to alternative mechanisms of cell death, resulting in more hepatocyte ballooning and liver fibrosis

the treated population, including patients with HCV genotype 3, compensated cirrhosis, and prior treatment experience (including treatment with SOF-based regimens), was the largest such patient group to date.[10, 11, 14] Among patients with GT3 infection and compensated cirrhosis, treatment with 12 and 16 weeks of RBV-free G/P yielded high rates of SVR12 in treatment-naive and treatment-experienced patients, respectively