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The elevation of Type I and Type II interferon-signaling pathways in cancer cells promotes their survival by evading NK cell killing (57) and suppressing effector T cell function by enhancing the accumulation of T regulatory (Treg) cells in the LNs (59)

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Furthermore, ML210 takes the form of diacylfuroxans, which transform into its corresponding -nitroketoxime, JKE-1674, within cells