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A decrease in phagocytosis, as well as ERK1/2 phosphorylation, was also seen in in response to dopamine (2 10 6 M) treatment of LPS-activated BV2 microglia, but no effect on the phagocytic activity of resting cells was observed, suggesting autocrine control of phagocytosis by dopamine signaling in activated myeloid cells (Fan et al., 2018)

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Because GHK-Cu interacts with regenerative, collagen-related, antioxidant-support, and tissue-maintenance pathways, observed responses may reflect broader physiological-resilience and recovery-focused signaling mechanisms associated with these pathways

Phua, T.J., Rogers, T.R., and Pallett, A.P