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The roles or mechanisms of 5-oxp associated with the regulation of plant tolerance to heat stress are not well understood, despite the importance of this natural metabolite, which is involved in key metabolic processes for stress defense
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Studies in the mouse model, which closely reproduces the human condition, have shown that hepatotoxicity is initiated by formation of a reactive metabolite N -acetyl- p -benzoquinone imine (NAPQI), which depletes cellular glutathione and forms protein adducts on mitochondrial proteins
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