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Acknowledgements We thank Annette Shadiack, Wilfredo Garcia, and Ramzey Odetalla for their valuable contributions to these studies Rights and permissions About this article Cite this article Diamond, L., Earle, D., Rosen, R

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NaV1.7 was once considered one of the most promising targets for non-opioid pain management after the discovery that individuals with loss-of-function mutations are insensitive to pain yet are otherwise healthy

ALKBH5 mechanistically reduced the stability of ACSL4 mRNA through m6A demethylation in a manner that was reliant on YTHDF1