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Synaptic density was increased in the cerebellum of Tsc1 -deficient Purkinje mice (13) and stem cells carrying ASD-related NLGN4 mutations (16)

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Related mode-of-action work has evaluated changes in body weight, fat mass, lean mass, and feeding behavior in controlled models, helping researchers explore how amylin pathway activation reshapes whole-body metabolic phenotypes [4]

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