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also found that Caveolin-1 deficiency inhibited the autophagic activity of TFCs and induced AKT /mTOR activation, which may be involved in the pathogenesis of HT [98]

doi: 10.3390/medsci7040056, PMID: [DOI] [PMC free article] [PubMed] [Google Scholar] 184.Botturi A, Ciappolino V, Delvecchio G, Boscutti A, Viscardi B, Brambilla P

The C-terminal rhodanese domain of CstB also displayed a similar fold with the human rhodanese (sulfurtransferase TSTD1) despite a meager 23 % sequence identity (Fig