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CYP2D6 may also contribute to the formation of NAPQI, albeit to a lesser extent than other P450 isozymes, and its activity may contribute to paracetamol toxicity, in particular, in extensive and ultrarapid metabolizers and when paracetamol is taken at very large doses.[18] The metabolism of paracetamol is an excellent example of toxication, because the metabolite NAPQI is primarily responsible for toxicity rather than paracetamol itself

Reduction in the rate of cell growth was also confirmed by MTT assay (Fig S3B)

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Mechanistically, their antioxidant activity stems from their ability to trap acyl chain-carrying peroxyl radicals in lipid bilayers