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Study endpoints include epithelial-barrier function, tight-junction protein expression, inflammatory signaling, and mucosal tissue remodeling in models of intestinal inflammation, increased permeability, and chemically induced gastrointestinal injury

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If the provision of some of these vitamins is insufficient, then a compensatory shift in pathways within OCM may occur (Reference Fernndez-Roig, Cavall-Busquets and Fernandez-Ballart12) but also it might have a negative impact on SAM synthesis with impairment of transmethylation reactions (Reference Bottiglieri11)

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