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The intrinsic agonist model of the ECD for the receptor activation is consistent with the ability of the N-terminally deleted hormone peptides to activate GLP-1R (Figure 9c and d) and the complete independence of GLP-1 and EX4 sequence for activation of the C347K/R mutated receptor (Figure 9e)

Thymosin Beta-4 (TB-4) plays a role in cell migration, wound repair, and anti-inflammatory signaling

Biosci Biotechnol Biochem 84(3):463470

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