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By outcompeting endogenous FOXO4 for p53 binding, FOXO4-DRI frees p53 to trigger apoptosis selectively in senescent cells

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While its activity is often associated with disease progression through the promotion of ferroptosis and oxidative damage, it also holds potential as a therapeutic target, with selective inhibition of CHAC1 offering possible benefits in conditions where its overexpression drives pathology

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