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Because IGF-1 is normally found at low levels in MetS and T2D, maybe because of insulin cessation to inhibit IGFBP-1 production by the liver and because of decreased liver IGF-1 secretion by insulin stimulation, as insulin resistance prevails in the liver

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FUT2 nonsecretor status links type 1 diabetes susceptibility and resistance to infection

It is distinguished in the research literature by its selectivity: published pre-clinical work reports it stimulates GH release without significantly elevating cortisol, prolactin, or ACTH, unlike earlier secretagogues in its class