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Although the exact mechanisms are still being investigated, the presence of dysregulated antibody responses in long COVID underscores the potential for antibodies to shift from protective to pathogenic roles

Human evidence is limited and largely cosmetic

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S-acetylglutathione has also been shown to decrease TNF-alpha, NF-kappa beta, and F-2 isoprostane.[4,9-12] Additionally, there is mounting evidence that intracellular glutathione levels in antigen-presenting cells (e.g macrophages) may influence the Th1/Th2 cytokine response pattern and promote a balanced immune reaction.*[10]