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Small-molecule compounds, including RAS-selective lethal 3 (RSL3) and erastin, promote ferroptosis, whereas proteins such as liproxstatin-1 (Lip-1), ferrostatin-1 (Fer-1), and the iron chelator deferoxamine (DFO) exert opposite effects (69)

A role for genetic has been demonstrated, with evidence of an increased risk of NAFL in patients with polymorphisms, like those associated to patatin-like phospholipase domain containing 3 (PNPLA3) rs738409, transmembrane 6 superfamily member 2 (TM6SF2) rs58542926 and membrane bound O-acyltransferase domain containing 7 (MBOAT7) rs641738 (4447)

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Cellular and Molecular Gastroenterology and Hepatology, 2 (3), 340-357