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The MTHFD2 enzyme is resistant to inhibition by NADH and so this reaction can contribute to significant mitochondrial NADH production under increased serine catabolism as well as during periods where the major NADH producing catabolic pathways are inhibited by increasing NADH levels

Emerging research highlights PPRs metabolic benefits, including enhanced insulin sensitivity and lipid metabolism regulation via AMPK activation and adipocyte differentiation modulation (21)

This lack of reproducibility is due to the distribution of extreme phenotypes (UM and PM) in these populations

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