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Kannengiesser and colleagues (2008) independently confirmed anti-inflammatory potential in two distinct murine IBD models, reducing colonic damage and inflammatory cytokine levels with the melanocortin-derived tripeptide

Both AOPPs and AGEs possess similar structures that induce comparable biological effects, and their accumulation in biological systems leads to similar clinical consequences

The key thing is that the bowel is kept clear as we kill off the overgrowth, so waste, dead bacteria and archaea and the toxins they produce are being removed from the body

Interestingly, previous research has shown upregulation of GPX4 at the transcriptome level during these stages [15]