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On the other hand, it is not clear whether FasL induces the formation of this complex, since it was reported that its activation was ROS-dependent and ligand-independent due to the inability of Fas agonists to activate caspase-8 and trigger cell membrane scrambling in mature RBCs [192]

These structural modifications reduce the molecules binding to IGF-binding proteins (IGFBPs) and extend its half-life relative to native IGF-1

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[1] [3] It should be avoided in patients with Leber's hereditary optic neuropathy