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Zha, X., Yang, B., Xia, G

Furthermore, in other disease models (such as A-induced nerve cell damage), highly expressed or activated VDAC1 has been clearly involved in ferroptosis, exacerbating cell death by inhibiting anti-ferroptosis pathways (such as GPX4, AMPK/mTOR, Wnt/-catenin) (149)

Experimental data indicate that increased PER1 expression may enhance the susceptibility of tumor cells to ionizing radiation, potentially affecting thresholds for radiation-induced cell death in in vitro and in vivo systems